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PARP inhibition protects mitochondria and reduces ROS production via PARP-1-ATF4-MKP-1-MAPK retrograde pathway - ScienceDirect

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ATF4 regulates arsenic trioxide-mediated NADPH oxidase, ER-mitochondrial crosstalk and apoptosis - ScienceDirect

Mitochondrial unfolded protein response: An emerging pathway in human diseases - ScienceDirect

Mitochondrial ROS in cancer: initiators, amplifiers or an Achilles' heel?

p53 at the crossroad between mitochondrial reactive oxygen species and necroptosis - ScienceDirect

Programmed necrotic cell death of macrophages: Focus on pyroptosis, necroptosis, and parthanatos - ScienceDirect

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ROS-induced DNA damage and PARP-1 are required for optimal induction of starvation-induced autophagy

New insights into vascular aging: Emerging role of mitochondria function - ScienceDirect

PDF) PARP inhibition induces Akt-mediated cytoprotective effects through the formation of a mitochondria-targeted phospho-ATM-NEMO-Akt-mTOR signalosome

PARP1 mediated PARylation contributes to myogenic progression and glucocorticoid transcriptional response

Nucleus-mitochondria positive feedback loop formed by ERK5 S496 phosphorylation-mediated poly (ADP-ribose) polymerase activation provokes persistent pro-inflammatory senescent phenotype and accelerates coronary atherosclerosis after chemo-radiation

Interplay between ADP-ribosyltransferases and essential cell signaling pathways controls cellular responses