GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F

Hepatic lipid profile in the LDLR knockout rats. Lipids were extracted

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

SETD2 deficiency accelerates sphingomyelin accumulation and promotes the development of renal cancer

SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair

Hepatic lipid profile in the LDLR knockout rats. Lipids were extracted

Fatty Acid Methyl ester GC-MS Mixture

RNA-seq analysis of 786-O and isogenic SETD2-deficient 38E and 38F

Quantitative analysis of fatty acids composition in the used cooking oil (UCO) by gas chromatography−mass spectrometry (GC–MS) - Abidin - 2013 - The Canadian Journal of Chemical Engineering - Wiley Online Library

GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F cells. (A)

SETD2 deficiency accelerates sphingomyelin accumulation and promotes the development of renal cancer

SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair

Gas chromatography/mass spectroscopy (GC/MS) spectrum of the silylated